File Download
  Links for fulltext
     (May Require Subscription)
  • Find via Find It@HKUL
Supplementary

Conference Paper: Regulatory role of miRNA-1246 and Wnt/β-catenin pathway interaction in CD133+ liver cancer stem cells-driven hepatocellular carcinoma

TitleRegulatory role of miRNA-1246 and Wnt/β-catenin pathway interaction in CD133+ liver cancer stem cells-driven hepatocellular carcinoma
Authors
Chai, SNg, KYTong, MGuan, XYMa, S
KeywordsMedical sciences
Oncology
Issue Date2014
PublisherAmerican Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/
Citation
The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1955 How to Cite?
AbstractHepatocellular carcinoma (HCC), the main type of liver cancer in human, is one of the most prevalent and deadly malignancies in the world. Despite advances in therapy, prognosis remains dismal, largely attributed to our limited understanding on information related to the progressive development of the disease, particularly in their cancer-initiating and stem cell-like properties. There is increasing evidence in recent years to support the role of miRNAs in the regulation of cancer stem cell (CSC) maintenance. However, miRNAs in hepatic CSCs remain understudied. Our group has previously identified a functional subset of liver CSCs marked by the CD133 phenotype (1-2). Utilizing a comprehensive transcriptome sequencing approach, we recently ...
DescriptionThis journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA
Persistent Identifierhttp://hdl.handle.net/10722/214998
ISSN
0008-5472
2021 Impact Factor: 13.312
2020 SCImago Journal Rankings: 4.103

 

DC FieldValueLanguage
dc.contributor.authorChai, S-
dc.contributor.authorNg, KY-
dc.contributor.authorTong, M-
dc.contributor.authorGuan, XY-
dc.contributor.authorMa, S-
dc.date.accessioned2015-08-21T12:17:48Z-
dc.date.available2015-08-21T12:17:48Z-
dc.date.issued2014-
dc.identifier.citationThe 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1955-
dc.identifier.issn0008-5472-
dc.identifier.urihttp://hdl.handle.net/10722/214998-
dc.descriptionThis journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA-
dc.description.abstractHepatocellular carcinoma (HCC), the main type of liver cancer in human, is one of the most prevalent and deadly malignancies in the world. Despite advances in therapy, prognosis remains dismal, largely attributed to our limited understanding on information related to the progressive development of the disease, particularly in their cancer-initiating and stem cell-like properties. There is increasing evidence in recent years to support the role of miRNAs in the regulation of cancer stem cell (CSC) maintenance. However, miRNAs in hepatic CSCs remain understudied. Our group has previously identified a functional subset of liver CSCs marked by the CD133 phenotype (1-2). Utilizing a comprehensive transcriptome sequencing approach, we recently ...-
dc.languageeng-
dc.publisherAmerican Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/-
dc.relation.ispartofCancer Research-
dc.subjectMedical sciences-
dc.subjectOncology-
dc.titleRegulatory role of miRNA-1246 and Wnt/β-catenin pathway interaction in CD133+ liver cancer stem cells-driven hepatocellular carcinoma-
dc.typeConference_Paper-
dc.identifier.emailNg, KY: jkyng@hku.hk-
dc.identifier.emailTong, M: caroltm@hku.hk-
dc.identifier.emailGuan, XY: xyguan@hkucc.hku.hk-
dc.identifier.emailMa, S: stefma@hku.hk-
dc.identifier.authorityGuan, XY=rp00454-
dc.identifier.authorityMa, S=rp00506-
dc.description.naturepostprint-
dc.identifier.hkuros248969-
dc.identifier.hkuros276954-
dc.identifier.volume74-
dc.identifier.issue19 suppl.-
dc.publisher.placeUnited States-
dc.identifier.issnl0008-5472-

Export via OAI-PMH Interface in XML Formats

OR

Export to Other Non-XML Formats