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Article: Gap junction inhibition by heptanol increases ventricular arrhythmogenicity by reducing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts

TitleGap junction inhibition by heptanol increases ventricular arrhythmogenicity by reducing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts
Authors
Tse, GYeo, JMTse, VKwan, SKJSun, B
KeywordsArrhythmia
Conduction
Conduction velocity
Gap junction
Refractory period
Repolarization
Wavelength
Issue Date2016
PublisherSpandidos Publications. The Journal's web site is located at http://www.spandidos-publications.com/mmr/
Citation
Molecular Medicine Reports, 2016, v. 14 n. 5, p. 4069-4074 How to Cite?
DOI: http://dx.doi.org/10.3892/mmr.2016.5738
Persistent Identifierhttp://hdl.handle.net/10722/227127
ISSN
1791-2997
2021 Impact Factor: 3.423
2020 SCImago Journal Rankings: 0.727
PubMed Central ID
PMC5101880
ISI Accession Number ID
WOS:000387241600009

 

DC FieldValueLanguage
dc.contributor.authorTse, G-
dc.contributor.authorYeo, JM-
dc.contributor.authorTse, V-
dc.contributor.authorKwan, SKJ-
dc.contributor.authorSun, B-
dc.date.accessioned2016-07-18T09:08:36Z-
dc.date.available2016-07-18T09:08:36Z-
dc.date.issued2016-
dc.identifier.citationMolecular Medicine Reports, 2016, v. 14 n. 5, p. 4069-4074-
dc.identifier.issn1791-2997-
dc.identifier.urihttp://hdl.handle.net/10722/227127-
dc.languageeng-
dc.publisherSpandidos Publications. The Journal's web site is located at http://www.spandidos-publications.com/mmr/-
dc.relation.ispartofMolecular Medicine Reports-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.subjectArrhythmia-
dc.subjectConduction-
dc.subjectConduction velocity-
dc.subjectGap junction-
dc.subjectRefractory period-
dc.subjectRepolarization-
dc.subjectWavelength-
dc.titleGap junction inhibition by heptanol increases ventricular arrhythmogenicity by reducing conduction velocity without affecting repolarization properties or myocardial refractoriness in Langendorff-perfused mouse hearts-
dc.typeArticle-
dc.identifier.emailTse, G: tseg@hku.hk-
dc.identifier.emailKwan, SKJ: jskkwan@hku.hk-
dc.identifier.authorityTse, G=rp02073-
dc.identifier.authorityKwan, SKJ=rp01868-
dc.description.naturepublished_or_final_version-
dc.identifier.doi10.3892/mmr.2016.5738-
dc.identifier.pmcidPMC5101880-
dc.identifier.scopuseid_2-s2.0-84992401781-
dc.identifier.hkuros259501-
dc.identifier.hkuros271392-
dc.identifier.volume14-
dc.identifier.issue5-
dc.identifier.spage4069-
dc.identifier.epage4074-
dc.identifier.isiWOS:000387241600009-
dc.publisher.placeGreece-
dc.identifier.issnl1791-2997-

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