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Article: Natural product celastrol suppressed macrophage M1 polarization against inflammation in diet-induced obese mice via regulating Nrf2/HO-1, MAP kinase and NF-κB pathways

TitleNatural product celastrol suppressed macrophage M1 polarization against inflammation in diet-induced obese mice via regulating Nrf2/HO-1, MAP kinase and NF-κB pathways
Authors
KeywordsAdipose tissue
Celastrol
Inflammation
Liver
Macrophage polarization
Obesity
Issue Date2017
PublisherImpact Journals LLC. The Journal's web site is located at http://www.impactaging.com
Citation
Aging, 2017, v. 9 n. 10, p. 2069-2082 How to Cite?
Persistent Identifierhttp://hdl.handle.net/10722/250022
ISSN
2021 Impact Factor: 5.955
2020 SCImago Journal Rankings: 1.473
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorLUO, D-
dc.contributor.authorGuo, Y-
dc.contributor.authorCHENG, Y-
dc.contributor.authorZhao, J-
dc.contributor.authorWang, Y-
dc.contributor.authorRong, J-
dc.date.accessioned2017-12-20T09:19:32Z-
dc.date.available2017-12-20T09:19:32Z-
dc.date.issued2017-
dc.identifier.citationAging, 2017, v. 9 n. 10, p. 2069-2082-
dc.identifier.issn1945-4589-
dc.identifier.urihttp://hdl.handle.net/10722/250022-
dc.languageeng-
dc.publisherImpact Journals LLC. The Journal's web site is located at http://www.impactaging.com-
dc.relation.ispartofAging-
dc.subjectAdipose tissue-
dc.subjectCelastrol-
dc.subjectInflammation-
dc.subjectLiver-
dc.subjectMacrophage polarization-
dc.subjectObesity-
dc.titleNatural product celastrol suppressed macrophage M1 polarization against inflammation in diet-induced obese mice via regulating Nrf2/HO-1, MAP kinase and NF-κB pathways-
dc.typeArticle-
dc.identifier.emailZhao, J: zhaojia7@hku.hk-
dc.identifier.emailWang, Y: yuwanghk@hku.hk-
dc.identifier.emailRong, J: jrong@hku.hk-
dc.identifier.authorityWang, Y=rp00239-
dc.identifier.authorityRong, J=rp00515-
dc.description.naturepublished_or_final_version-
dc.identifier.doi10.18632/aging.101302-
dc.identifier.scopuseid_2-s2.0-85032733865-
dc.identifier.hkuros283831-
dc.identifier.volume9-
dc.identifier.issue10-
dc.identifier.spage2069-
dc.identifier.epage2082-
dc.identifier.isiWOS:000414179600011-
dc.publisher.placeUnited States-
dc.identifier.issnl1945-4589-

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