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Article: Vasopressin-independent mechanisms in controlling water homeostasis

TitleVasopressin-independent mechanisms in controlling water homeostasis
Authors
Cheng, CYYChu, JYSChow, BKC
Issue Date2009
PublisherSociety for Endocrinology. The Journal's web site is located at http://jme.endocrinology-journals.org/
Citation
Journal Of Molecular Endocrinology, 2009, v. 43 n. 3, p. 81-92 How to Cite?
DOI: http://dx.doi.org/10.1677/JME-08-0123
AbstractThe maintenance of body water homeostasis depends on the balance between water intake and water excretion. In the kidney, vasopressin (Vp) is a critical regulator of water homeostasis by controlling the insertion of aquaporin 2 (AQP2) onto the apical membrane of the collecting duct principal cells in the short term and regulating the gene expression of AQP2 in the long term. A growing body of evidence from both in vitro and in vivo studies demonstrated that both secretin and oxytocin are involved as Vp-independent mechanisms regulating the renal water reabsorption process, including the translocation and expression of AQP2. This review focuses on how these two hormones are potentially involved as Vp-independent mechanisms in controlling water homeostasis. © 2009 Society for Endocrinology.
Persistent Identifierhttp://hdl.handle.net/10722/89196
ISSN
0952-5041
2021 Impact Factor: 4.869
2020 SCImago Journal Rankings: 1.452
ISI Accession Number ID
WOS:000270477400001
Funding AgencyGrant Number
Research Grant Council HKU7501/05 M
7566/06
Funding Information:

This work was supported by a research grant from Research Grant Council HKU 7501/05 M and 7566/06 M to BKC Chow.

References
References in Scopus

 

DC FieldValueLanguage
dc.contributor.authorCheng, CYYen_HK
dc.contributor.authorChu, JYSen_HK
dc.contributor.authorChow, BKCen_HK
dc.date.accessioned2010-09-06T09:53:44Z-
dc.date.available2010-09-06T09:53:44Z-
dc.date.issued2009en_HK
dc.identifier.citationJournal Of Molecular Endocrinology, 2009, v. 43 n. 3, p. 81-92en_HK
dc.identifier.issn0952-5041en_HK
dc.identifier.urihttp://hdl.handle.net/10722/89196-
dc.description.abstractThe maintenance of body water homeostasis depends on the balance between water intake and water excretion. In the kidney, vasopressin (Vp) is a critical regulator of water homeostasis by controlling the insertion of aquaporin 2 (AQP2) onto the apical membrane of the collecting duct principal cells in the short term and regulating the gene expression of AQP2 in the long term. A growing body of evidence from both in vitro and in vivo studies demonstrated that both secretin and oxytocin are involved as Vp-independent mechanisms regulating the renal water reabsorption process, including the translocation and expression of AQP2. This review focuses on how these two hormones are potentially involved as Vp-independent mechanisms in controlling water homeostasis. © 2009 Society for Endocrinology.en_HK
dc.languageengen_HK
dc.publisherSociety for Endocrinology. The Journal's web site is located at http://jme.endocrinology-journals.org/en_HK
dc.relation.ispartofJournal of Molecular Endocrinologyen_HK
dc.rightsJournal of Molecular Endocrinology. Copyright © Society for Endocrinology.en_HK
dc.titleVasopressin-independent mechanisms in controlling water homeostasisen_HK
dc.typeArticleen_HK
dc.identifier.openurlhttp://library.hku.hk:4550/resserv?sid=HKU:IR&issn=0952-5041&volume=43&spage=81&epage=92&date=2009&atitle=Vasopressin-independent+mechanisms+in+controlling+water+homeostasisen_HK
dc.identifier.emailChu, JYS: hitan@graduate.hku.hken_HK
dc.identifier.emailChow, BKC: bkcc@hku.hken_HK
dc.identifier.authorityChu, JYS=rp00684en_HK
dc.identifier.authorityChow, BKC=rp00681en_HK
dc.description.naturelink_to_OA_fulltext-
dc.identifier.doi10.1677/JME-08-0123en_HK
dc.identifier.pmid19318428-
dc.identifier.scopuseid_2-s2.0-70350738250en_HK
dc.identifier.hkuros162709en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-70350738250&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume43en_HK
dc.identifier.issue3en_HK
dc.identifier.spage81en_HK
dc.identifier.epage92en_HK
dc.identifier.isiWOS:000270477400001-
dc.publisher.placeUnited Kingdomen_HK
dc.identifier.scopusauthoridCheng, CYY=35423534900en_HK
dc.identifier.scopusauthoridChu, JYS=34975209300en_HK
dc.identifier.scopusauthoridChow, BKC=7102826193en_HK
dc.identifier.citeulike4498902-
dc.identifier.issnl0952-5041-

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