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postgraduate thesis: ErbB receptor modulation by the Notch pathway as a means to fate commitment in bone marrow-derived Schwann cells

TitleErbB receptor modulation by the Notch pathway as a means to fate commitment in bone marrow-derived Schwann cells
Authors
Issue Date2011
PublisherThe University of Hong Kong (Pokfulam, Hong Kong)
Citation
Shea, K. G. [佘嘉翰]. (2011). ErbB receptor modulation by the Notch pathway as a means to fate commitment in bone marrow-derived Schwann cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4608304
DegreeDoctor of Philosophy
SubjectNeuroglia.
Epidermal growth factor - Receptors.
Cellular signal transduction.
Dept/ProgramBiochemistry
Persistent Identifierhttp://hdl.handle.net/10722/134811
HKU Library Item IDb4608304

 

DC FieldValueLanguage
dc.contributor.authorShea, Ka-hon, Graham-
dc.contributor.author佘嘉翰zh_HK
dc.date.accessioned2011-07-19T01:08:30Z-
dc.date.available2011-07-19T01:08:30Z-
dc.date.issued2011-
dc.identifier.citationShea, K. G. [佘嘉翰]. (2011). ErbB receptor modulation by the Notch pathway as a means to fate commitment in bone marrow-derived Schwann cells. (Thesis). University of Hong Kong, Pokfulam, Hong Kong SAR. Retrieved from http://dx.doi.org/10.5353/th_b4608304-
dc.identifier.urihttp://hdl.handle.net/10722/134811-
dc.languageeng-
dc.publisherThe University of Hong Kong (Pokfulam, Hong Kong)-
dc.relation.ispartofHKU Theses Online (HKUTO)-
dc.rightsThe author retains all proprietary rights, (such as patent rights) and the right to use in future works.-
dc.source.urihttp://hub.hku.hk/bib/B46083042-
dc.subject.lcshNeuroglia.-
dc.subject.lcshEpidermal growth factor - Receptors.-
dc.subject.lcshCellular signal transduction.-
dc.titleErbB receptor modulation by the Notch pathway as a means to fate commitment in bone marrow-derived Schwann cells-
dc.typePG_Thesis-
dc.identifier.hkulb4608304-
dc.description.thesisnameDoctor of Philosophy-
dc.description.thesislevelDoctoral-
dc.description.thesisdisciplineBiochemistry-
dc.description.natureabstract-
dc.identifier.doi10.5353/th_b4608304-
dc.identifier.mmsid991031913549703414-

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