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Conference Paper: Deficiency of EP4 reeptor on bone marrow-derived cells boosted inflammation and abdominal aortic aneurysm formation induced by Angiotensin II in hyperlipidemic mice

TitleDeficiency of EP4 reeptor on bone marrow-derived cells boosted inflammation and abdominal aortic aneurysm formation induced by Angiotensin II in hyperlipidemic mice
Authors
Tang, EHCShvartz, EShimizu, KRocha, VZZheng, CFukuda, DShi, GPSukhova, GLibby, P
KeywordsCardiovascular disease
Issue Date2010
PublisherHong Kong College of Cardiology. The Journal's web site is located at http://www.hkcchk.com/journals.php#3
Citation
The 14th Annual Scientific Meetingof the Institute of Cardiovascular Science and Medicine, Hong Kong, China, 18 December 2010. In Journal of the Hong Kong College of Cardiology, 2010, v. 18 n. 2, p. 63, abstract no. OC2 How to Cite?
AbstractOBJECTIVE: Chronic inflammation during abdominal aortic aneurysm (AAA) formation contributes to remodeling and eventual weakening of the vessel wall. Prostaglandin E2 (PGE2), through activation of its receptor EP4, can mute inflammation. Whether EP4 participates directly in the pathogenesis of aneurysm remains unknown. We tested the hypothesis that ...
DescriptionThis journal issue is proceedings of ICSM 2010
Abstracts for Oral Communications
Persistent Identifierhttp://hdl.handle.net/10722/136061
ISSN
1027-7811
2020 SCImago Journal Rankings: 0.105

 

DC FieldValueLanguage
dc.contributor.authorTang, EHCen_US
dc.contributor.authorShvartz, Een_US
dc.contributor.authorShimizu, Ken_US
dc.contributor.authorRocha, VZen_US
dc.contributor.authorZheng, Cen_US
dc.contributor.authorFukuda, Den_US
dc.contributor.authorShi, GPen_US
dc.contributor.authorSukhova, Gen_US
dc.contributor.authorLibby, Pen_US
dc.date.accessioned2011-07-27T02:02:17Z-
dc.date.available2011-07-27T02:02:17Z-
dc.date.issued2010en_US
dc.identifier.citationThe 14th Annual Scientific Meetingof the Institute of Cardiovascular Science and Medicine, Hong Kong, China, 18 December 2010. In Journal of the Hong Kong College of Cardiology, 2010, v. 18 n. 2, p. 63, abstract no. OC2en_US
dc.identifier.issn1027-7811-
dc.identifier.urihttp://hdl.handle.net/10722/136061-
dc.descriptionThis journal issue is proceedings of ICSM 2010-
dc.descriptionAbstracts for Oral Communications-
dc.description.abstractOBJECTIVE: Chronic inflammation during abdominal aortic aneurysm (AAA) formation contributes to remodeling and eventual weakening of the vessel wall. Prostaglandin E2 (PGE2), through activation of its receptor EP4, can mute inflammation. Whether EP4 participates directly in the pathogenesis of aneurysm remains unknown. We tested the hypothesis that ...-
dc.languageengen_US
dc.publisherHong Kong College of Cardiology. The Journal's web site is located at http://www.hkcchk.com/journals.php#3-
dc.relation.ispartofJournal of the Hong Kong College of Cardiologyen_US
dc.subjectCardiovascular disease-
dc.titleDeficiency of EP4 reeptor on bone marrow-derived cells boosted inflammation and abdominal aortic aneurysm formation induced by Angiotensin II in hyperlipidemic miceen_US
dc.typeConference_Paperen_US
dc.identifier.emailTang, EHC: evatang1@hku.hken_US
dc.identifier.authorityTang, EHC=rp01382en_US
dc.description.naturelink_to_OA_fulltext-
dc.identifier.hkuros186289en_US
dc.identifier.volume18en_US
dc.identifier.issue2en_US
dc.identifier.spage63, abstract no. OC2en_US
dc.identifier.epage63, abstract no. OC2en_US
dc.publisher.placeHong Kong-
dc.description.otherThe 14th Annual Scientific Meetingof the Institute of Cardiovascular Science and Medicine, Hong Kong, China, 18 December 2010. In Journal of the Hong Kong College of Cardiology, 2010, v. 18 n. 2, p. 63, abstract no. OC2-
dc.identifier.issnl1027-7811-

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