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Conference Paper: Mortalin-p53 interaction in cancer cells is stress dependent and constitutes a novel target for liver cancer therapy

TitleMortalin-p53 interaction in cancer cells is stress dependent and constitutes a novel target for liver cancer therapy
Authors
Lu, WJLee, NPKaul, SCPoon, RTWadhwa, RLuk, JM
KeywordsMedical sciences
Gastroenterology
Issue Date2011
PublisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/jhep
Citation
The 46th Annual Meeting of the European Association for the Study of the Liver ( International Liver Congress™ 2011), Berlin, Germany, 30 March-3 April 2011. In Journal of Hepatology, 2011, v. 54 suppl. 1, p. S272, abstract no. 676 How to Cite?
DOI: http://dx.doi.org/10.1016/S0168-8278(11)60678-8
AbstractBACKGROUND AND AIMS: The mortality rate of HCC is high due to tumor recurrence and lack of effective treatment. By proteomics analysis of matched tumor and non-tumor tissues, mortalin was identified as a marker for hepatocellular carcinoma (HCC) metastasis and recurrence, suggesting its tight link in HCC development and recurrence. The aim of this study is to examine the role of mortalin in hepatocarcinogenesis. METHODS: The mortalin expression ...
DescriptionThis journal suppl. entitled: The International Liver Congress™ 2011 Abstract Book 46 annual meeting of the European Association for the Study of the Liver
Poster Abstracts
Persistent Identifierhttp://hdl.handle.net/10722/137900
ISSN
0168-8278
2023 Impact Factor: 26.8
2023 SCImago Journal Rankings: 9.857
ISI Accession Number ID
WOS:000297625601254

 

DC FieldValueLanguage
dc.contributor.authorLu, WJ-
dc.contributor.authorLee, NP-
dc.contributor.authorKaul, SC-
dc.contributor.authorPoon, RT-
dc.contributor.authorWadhwa, R-
dc.contributor.authorLuk, JM-
dc.date.accessioned2011-08-26T14:36:39Z-
dc.date.available2011-08-26T14:36:39Z-
dc.date.issued2011-
dc.identifier.citationThe 46th Annual Meeting of the European Association for the Study of the Liver ( International Liver Congress™ 2011), Berlin, Germany, 30 March-3 April 2011. In Journal of Hepatology, 2011, v. 54 suppl. 1, p. S272, abstract no. 676-
dc.identifier.issn0168-8278-
dc.identifier.urihttp://hdl.handle.net/10722/137900-
dc.descriptionThis journal suppl. entitled: The International Liver Congress™ 2011 Abstract Book 46 annual meeting of the European Association for the Study of the Liver-
dc.descriptionPoster Abstracts-
dc.description.abstractBACKGROUND AND AIMS: The mortality rate of HCC is high due to tumor recurrence and lack of effective treatment. By proteomics analysis of matched tumor and non-tumor tissues, mortalin was identified as a marker for hepatocellular carcinoma (HCC) metastasis and recurrence, suggesting its tight link in HCC development and recurrence. The aim of this study is to examine the role of mortalin in hepatocarcinogenesis. METHODS: The mortalin expression ...-
dc.languageeng-
dc.publisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/jhep-
dc.relation.ispartofJournal of Hepatology-
dc.rightsNOTICE: this is the author’s version of a work that was accepted for publication in Journal of Hepatology. Changes resulting from the publishing process, such as peer review, editing, corrections, structural formatting, and other quality control mechanisms may not be reflected in this document. Changes may have been made to this work since it was submitted for publication. A definitive version was subsequently published in Journal of Hepatology, v. 54, suppl. 1 (March 2011). DOI: 10.1016/S0168-8278(11)60678-8-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.subjectMedical sciences-
dc.subjectGastroenterology-
dc.titleMortalin-p53 interaction in cancer cells is stress dependent and constitutes a novel target for liver cancer therapy-
dc.typeConference_Paper-
dc.identifier.emailLee, NP: nikkilee@hku.hk-
dc.identifier.emailPoon, RT: poontp@hku.hk-
dc.identifier.emailLuk, JM: jmluk@hkucc.hku.hk-
dc.identifier.authorityLee, NP=rp00263-
dc.identifier.authorityPoon, RT=rp00446-
dc.identifier.authorityLuk, JM=rp00349-
dc.description.naturepostprint-
dc.identifier.doi10.1016/S0168-8278(11)60678-8-
dc.identifier.hkuros190046-
dc.identifier.volume54-
dc.identifier.issuesuppl. 1-
dc.identifier.spageS272, abstract no. 676-
dc.identifier.epageS272, abstract no. 676-
dc.identifier.isiWOS:000297625601254-
dc.publisher.placeThe Netherlands-
dc.identifier.issnl0168-8278-

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