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Article: Sudden death associated with bloodless aortic dissection

TitleSudden death associated with bloodless aortic dissection
Authors
KeywordsAortic dissection
Bloodless
Cystic medionecrosis
Sudden death
Issue Date1993
PublisherElsevier Ireland Ltd. The Journal's web site is located at http://www.elsevier.com/locate/forsciint
Citation
Forensic Science International, 1993, v. 59 n. 2, p. 149-155 How to Cite?
AbstractWe report a case of rapidly fatal aortic dissection which was unusual because no intimal tear was present and no blood was present in the dissected aortic wall, although the dissection involved the entire aorta. Death was considered to be due to acute myocardial ischaemia from dissection involving the left coronary artery. Prominent cystic medial necrosis was present. There was no history of hypertension and only histological evidence of left ventricular hypertrophy. No other clinical or pathologic states associated with aortic dissection were present. Although cystic medionecrosis was the probable cause in this case, it is present in only a small proportion of patients in the absence of Marfan's syndrome, and more recently investigated defects in the composition of aortic wall connective tissue components may be more relevant factors in the causation of many cases of aortic dissection.
Persistent Identifierhttp://hdl.handle.net/10722/147958
ISSN
2023 Impact Factor: 2.2
2023 SCImago Journal Rankings: 0.750
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorDickens, Pen_US
dc.contributor.authorKhoo, USen_US
dc.date.accessioned2012-05-29T06:10:06Z-
dc.date.available2012-05-29T06:10:06Z-
dc.date.issued1993en_US
dc.identifier.citationForensic Science International, 1993, v. 59 n. 2, p. 149-155en_US
dc.identifier.issn0379-0738en_US
dc.identifier.urihttp://hdl.handle.net/10722/147958-
dc.description.abstractWe report a case of rapidly fatal aortic dissection which was unusual because no intimal tear was present and no blood was present in the dissected aortic wall, although the dissection involved the entire aorta. Death was considered to be due to acute myocardial ischaemia from dissection involving the left coronary artery. Prominent cystic medial necrosis was present. There was no history of hypertension and only histological evidence of left ventricular hypertrophy. No other clinical or pathologic states associated with aortic dissection were present. Although cystic medionecrosis was the probable cause in this case, it is present in only a small proportion of patients in the absence of Marfan's syndrome, and more recently investigated defects in the composition of aortic wall connective tissue components may be more relevant factors in the causation of many cases of aortic dissection.en_US
dc.languageengen_US
dc.publisherElsevier Ireland Ltd. The Journal's web site is located at http://www.elsevier.com/locate/forsciinten_US
dc.relation.ispartofForensic Science Internationalen_US
dc.subjectAortic dissection-
dc.subjectBloodless-
dc.subjectCystic medionecrosis-
dc.subjectSudden death-
dc.subject.meshAneurysm, Dissecting - Classification - Diagnosis - Epidemiology - Etiologyen_US
dc.subject.meshAorta - Pathologyen_US
dc.subject.meshAortic Aneurysm - Classification - Diagnosis - Epidemiology - Etiologyen_US
dc.subject.meshBiopsyen_US
dc.subject.meshCausalityen_US
dc.subject.meshDeath, Sudden - Etiologyen_US
dc.subject.meshEchocardiographyen_US
dc.subject.meshElectrocardiographyen_US
dc.subject.meshHumansen_US
dc.subject.meshMaleen_US
dc.subject.meshMiddle Ageden_US
dc.subject.meshNecrosisen_US
dc.subject.meshTunica Media - Pathologyen_US
dc.titleSudden death associated with bloodless aortic dissectionen_US
dc.typeArticleen_US
dc.identifier.emailKhoo, US:uskhoo@hkucc.hku.hken_US
dc.identifier.authorityKhoo, US=rp00362en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1016/0379-0738(93)90153-2en_US
dc.identifier.pmid8330807-
dc.identifier.scopuseid_2-s2.0-0027238335en_US
dc.identifier.volume59en_US
dc.identifier.issue2en_US
dc.identifier.spage149en_US
dc.identifier.epage155en_US
dc.identifier.isiWOS:A1993LG93700007-
dc.publisher.placeIrelanden_US
dc.identifier.issnl0379-0738-

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