File Download
There are no files associated with this item.
Links for fulltext
(May Require Subscription)
- Publisher Website: 10.1007/s00277-001-0423-6
- Scopus: eid_2-s2.0-0036943733
- PMID: 11904745
- WOS: WOS:000175035100009
- Find via
Supplementary
- Citations:
- Appears in Collections:
Article: Fatal diffuse alveolar damage complicating acute myeloid leukemia with abnormal eosinophils and trisomy X
Title | Fatal diffuse alveolar damage complicating acute myeloid leukemia with abnormal eosinophils and trisomy X |
---|---|
Authors | |
Keywords | Abnormal eosinophils AML Cytogenetics Diffuse alveolar damage Trisomy X |
Issue Date | 2002 |
Publisher | Springer Verlag. The Journal's web site is located at http://link.springer.de/link/service/journals/00277/index.htm |
Citation | Annals Of Hematology, 2002, v. 81 n. 3, p. 167-169 How to Cite? |
Abstract | We describe a case of acute myeloid leukemia (AML) with abnormal eosinophils in a 44-year-old Chinese woman that was complicated by diffuse alveolar damage (DAD) and pulmonary hemorrhage (PH) shortly after induction chemotherapy. Cytogenetic study of bone marrow cells at diagnosis showed a rare aberration of trisomy X (+X) as the sole acquired karyotypic abnormality. We speculate that tissue damage by cellular constituents of the abnormal eosinophils that were released on cell lysis after chemotherapy might be etiologically linked to the occurrence of fatal pulmonary complications. |
Persistent Identifier | http://hdl.handle.net/10722/148303 |
ISSN | 2023 Impact Factor: 3.0 2023 SCImago Journal Rankings: 0.912 |
ISI Accession Number ID | |
References |
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Wan, TSK | en_US |
dc.contributor.author | Yip, SF | en_US |
dc.contributor.author | Yeung, YM | en_US |
dc.contributor.author | Chan, LC | en_US |
dc.contributor.author | Ma, SK | en_US |
dc.date.accessioned | 2012-05-29T06:12:06Z | - |
dc.date.available | 2012-05-29T06:12:06Z | - |
dc.date.issued | 2002 | en_US |
dc.identifier.citation | Annals Of Hematology, 2002, v. 81 n. 3, p. 167-169 | en_US |
dc.identifier.issn | 0939-5555 | en_US |
dc.identifier.uri | http://hdl.handle.net/10722/148303 | - |
dc.description.abstract | We describe a case of acute myeloid leukemia (AML) with abnormal eosinophils in a 44-year-old Chinese woman that was complicated by diffuse alveolar damage (DAD) and pulmonary hemorrhage (PH) shortly after induction chemotherapy. Cytogenetic study of bone marrow cells at diagnosis showed a rare aberration of trisomy X (+X) as the sole acquired karyotypic abnormality. We speculate that tissue damage by cellular constituents of the abnormal eosinophils that were released on cell lysis after chemotherapy might be etiologically linked to the occurrence of fatal pulmonary complications. | en_US |
dc.language | eng | en_US |
dc.publisher | Springer Verlag. The Journal's web site is located at http://link.springer.de/link/service/journals/00277/index.htm | en_US |
dc.relation.ispartof | Annals of Hematology | en_US |
dc.subject | Abnormal eosinophils | - |
dc.subject | AML | - |
dc.subject | Cytogenetics | - |
dc.subject | Diffuse alveolar damage | - |
dc.subject | Trisomy X | - |
dc.subject.mesh | Acute Disease | en_US |
dc.subject.mesh | Adult | en_US |
dc.subject.mesh | Eosinophils - Pathology | en_US |
dc.subject.mesh | Fatal Outcome | en_US |
dc.subject.mesh | Female | en_US |
dc.subject.mesh | Hemorrhage - Complications | en_US |
dc.subject.mesh | Humans | en_US |
dc.subject.mesh | Leukemia, Myeloid - Complications - Genetics - Pathology | en_US |
dc.subject.mesh | Lung Diseases - Complications | en_US |
dc.subject.mesh | Pulmonary Alveoli - Pathology | en_US |
dc.subject.mesh | Trisomy - Genetics | en_US |
dc.subject.mesh | X Chromosome - Genetics | en_US |
dc.title | Fatal diffuse alveolar damage complicating acute myeloid leukemia with abnormal eosinophils and trisomy X | en_US |
dc.type | Article | en_US |
dc.identifier.email | Chan, LC:chanlc@hkucc.hku.hk | en_US |
dc.identifier.authority | Chan, LC=rp00373 | en_US |
dc.description.nature | link_to_subscribed_fulltext | en_US |
dc.identifier.doi | 10.1007/s00277-001-0423-6 | en_US |
dc.identifier.pmid | 11904745 | - |
dc.identifier.scopus | eid_2-s2.0-0036943733 | en_US |
dc.relation.references | http://www.scopus.com/mlt/select.url?eid=2-s2.0-0036943733&selection=ref&src=s&origin=recordpage | en_US |
dc.identifier.volume | 81 | en_US |
dc.identifier.issue | 3 | en_US |
dc.identifier.spage | 167 | en_US |
dc.identifier.epage | 169 | en_US |
dc.identifier.isi | WOS:000175035100009 | - |
dc.publisher.place | Germany | en_US |
dc.identifier.issnl | 0939-5555 | - |