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Article: MyelomA genetics international consortium

TitleMyelomA genetics international consortium
Authors
Morgan, GJohnsen, HEGoldschmidt, HPalumbo, ACavo, MSonneveld, PMiguel, JSChim, CSBrowne, PEinsele, HWaage, ATuresson, ISpencer, AHajek, RLudwig, HHemminki, KHoulston, R
KeywordsConsortium
Etiology
Genetics
Multiple Myeloma
Issue Date2012
PublisherInforma Healthcare. The Journal's web site is located at http://informahealthcare.com/loi/lal
Citation
Leukemia And Lymphoma, 2012, v. 53 n. 5, p. 796-800 How to Cite?
DOI: http://dx.doi.org/10.3109/10428194.2011.639881
AbstractWhile the etiology of multiple myeloma (MM) is largely unknown, evidence for an inherited genetic susceptibility is provided by the two-fold increased risk of the disease seen in first-degree relatives of cases of MM. It is likely that part of this heritable risk is a consequence of the co-inheritance of low-risk genetic variants. The accumulated experience to date in identifying risk variants for other tumors has highlighted difficulties in conducting statistically and methodologically rigorous studies. The MyelomA Genetics International Consortium (MAGIC) includes 16 research groups in Europe, Asia, Australasia, the Middle East and the Americas engaged in studying the genetics of MM. The first goal of MAGIC is to identify and characterize common genetic variants for MM through association-based analyses. Here, we review the rationale for identifying genetic risk variants for MM and our proposed strategy for establishing MAGIC. © 2012 Informa UK, Ltd.
Persistent Identifierhttp://hdl.handle.net/10722/163478
ISSN
1042-8194
2023 Impact Factor: 2.2
2023 SCImago Journal Rankings: 0.790
ISI Accession Number ID
WOS:000303070900010
References
References in Scopus

 

DC FieldValueLanguage
dc.contributor.authorMorgan, Gen_US
dc.contributor.authorJohnsen, HEen_US
dc.contributor.authorGoldschmidt, Hen_US
dc.contributor.authorPalumbo, Aen_US
dc.contributor.authorCavo, Men_US
dc.contributor.authorSonneveld, Pen_US
dc.contributor.authorMiguel, JSen_US
dc.contributor.authorChim, CSen_US
dc.contributor.authorBrowne, Pen_US
dc.contributor.authorEinsele, Hen_US
dc.contributor.authorWaage, Aen_US
dc.contributor.authorTuresson, Ien_US
dc.contributor.authorSpencer, Aen_US
dc.contributor.authorHajek, Ren_US
dc.contributor.authorLudwig, Hen_US
dc.contributor.authorHemminki, Ken_US
dc.contributor.authorHoulston, Ren_US
dc.date.accessioned2012-09-05T05:31:49Z-
dc.date.available2012-09-05T05:31:49Z-
dc.date.issued2012en_US
dc.identifier.citationLeukemia And Lymphoma, 2012, v. 53 n. 5, p. 796-800en_US
dc.identifier.issn1042-8194en_US
dc.identifier.urihttp://hdl.handle.net/10722/163478-
dc.description.abstractWhile the etiology of multiple myeloma (MM) is largely unknown, evidence for an inherited genetic susceptibility is provided by the two-fold increased risk of the disease seen in first-degree relatives of cases of MM. It is likely that part of this heritable risk is a consequence of the co-inheritance of low-risk genetic variants. The accumulated experience to date in identifying risk variants for other tumors has highlighted difficulties in conducting statistically and methodologically rigorous studies. The MyelomA Genetics International Consortium (MAGIC) includes 16 research groups in Europe, Asia, Australasia, the Middle East and the Americas engaged in studying the genetics of MM. The first goal of MAGIC is to identify and characterize common genetic variants for MM through association-based analyses. Here, we review the rationale for identifying genetic risk variants for MM and our proposed strategy for establishing MAGIC. © 2012 Informa UK, Ltd.en_US
dc.languageengen_US
dc.publisherInforma Healthcare. The Journal's web site is located at http://informahealthcare.com/loi/lalen_US
dc.relation.ispartofLeukemia and Lymphomaen_US
dc.subjectConsortiumen_US
dc.subjectEtiologyen_US
dc.subjectGeneticsen_US
dc.subjectMultiple Myelomaen_US
dc.titleMyelomA genetics international consortiumen_US
dc.typeArticleen_US
dc.identifier.emailChim, CS:jcschim@hku.hken_US
dc.identifier.authorityChim, CS=rp00408en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.3109/10428194.2011.639881en_US
dc.identifier.pmid22080755-
dc.identifier.scopuseid_2-s2.0-84859972113en_US
dc.identifier.hkuros209923-
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-84859972113&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume53en_US
dc.identifier.issue5en_US
dc.identifier.spage796en_US
dc.identifier.epage800en_US
dc.identifier.isiWOS:000303070900010-
dc.publisher.placeUnited Kingdomen_US
dc.identifier.scopusauthoridMorgan, G=36038178500en_US
dc.identifier.scopusauthoridJohnsen, HE=7103033331en_US
dc.identifier.scopusauthoridGoldschmidt, H=26643000200en_US
dc.identifier.scopusauthoridPalumbo, A=7103361944en_US
dc.identifier.scopusauthoridCavo, M=7005399620en_US
dc.identifier.scopusauthoridSonneveld, P=7005618857en_US
dc.identifier.scopusauthoridMiguel, JS=7004412126en_US
dc.identifier.scopusauthoridChim, CS=7004597253en_US
dc.identifier.scopusauthoridBrowne, P=55192867900en_US
dc.identifier.scopusauthoridEinsele, H=55159011600en_US
dc.identifier.scopusauthoridWaage, A=7007141045en_US
dc.identifier.scopusauthoridTuresson, I=7005867886en_US
dc.identifier.scopusauthoridSpencer, A=55159070000en_US
dc.identifier.scopusauthoridHajek, R=55150911900en_US
dc.identifier.scopusauthoridLudwig, H=7201526238en_US
dc.identifier.scopusauthoridHemminki, K=36044571800en_US
dc.identifier.scopusauthoridHoulston, R=34569730700en_US
dc.identifier.issnl1026-8022-

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