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Article: Fetal origins of reduced arterial distensibility in the donor twin in twin-twin transfusion syndrome

TitleFetal origins of reduced arterial distensibility in the donor twin in twin-twin transfusion syndrome
Authors
Cheung, YFTaylor, MJOFisk, NMRedington, ANGardiner, HM
Issue Date2000
PublisherThe Lancet Publishing Group. The Journal's web site is located at http://www.elsevier.com/locate/lancet
Citation
Lancet, 2000, v. 355 n. 9210, p. 1157-1158 How to Cite?
DOI: http://dx.doi.org/10.1016/S0140-6736(00)02068-7
AbstractTwin-twin transfusion syndrome permits investigation of vascular programming independent of genetic influence. Arterial distensibiity was lower in the donor twin during infancy, implying the intrauterine vascular remodelling might result in raised cardiac afterload and could influence later cardiovascular health.
Persistent Identifierhttp://hdl.handle.net/10722/170305
ISSN
0140-6736
2023 Impact Factor: 98.4
2023 SCImago Journal Rankings: 12.113
ISI Accession Number ID
WOS:000086380900017
References
References in Scopus

 

DC FieldValueLanguage
dc.contributor.authorCheung, YFen_US
dc.contributor.authorTaylor, MJOen_US
dc.contributor.authorFisk, NMen_US
dc.contributor.authorRedington, ANen_US
dc.contributor.authorGardiner, HMen_US
dc.date.accessioned2012-10-30T06:07:22Z-
dc.date.available2012-10-30T06:07:22Z-
dc.date.issued2000en_US
dc.identifier.citationLancet, 2000, v. 355 n. 9210, p. 1157-1158en_US
dc.identifier.issn0140-6736en_US
dc.identifier.urihttp://hdl.handle.net/10722/170305-
dc.description.abstractTwin-twin transfusion syndrome permits investigation of vascular programming independent of genetic influence. Arterial distensibiity was lower in the donor twin during infancy, implying the intrauterine vascular remodelling might result in raised cardiac afterload and could influence later cardiovascular health.en_US
dc.languageengen_US
dc.publisherThe Lancet Publishing Group. The Journal's web site is located at http://www.elsevier.com/locate/lanceten_US
dc.relation.ispartofLanceten_US
dc.subject.meshBlood Flow Velocityen_US
dc.subject.meshBlood Pressureen_US
dc.subject.meshBrachial Artery - Physiopathologyen_US
dc.subject.meshDiseases In Twinsen_US
dc.subject.meshFemaleen_US
dc.subject.meshFetofetal Transfusion - Physiopathologyen_US
dc.subject.meshHumansen_US
dc.subject.meshInfanten_US
dc.subject.meshInfant, Newbornen_US
dc.subject.meshPhotoplethysmographyen_US
dc.subject.meshPregnancyen_US
dc.subject.meshPulsatile Flow - Physiologyen_US
dc.subject.meshRadial Artery - Physiopathologyen_US
dc.subject.meshTwins, Dizygoticen_US
dc.subject.meshTwins, Monozygoticen_US
dc.subject.meshVascular Resistance - Physiologyen_US
dc.titleFetal origins of reduced arterial distensibility in the donor twin in twin-twin transfusion syndromeen_US
dc.typeArticleen_US
dc.identifier.emailCheung, YF:xfcheung@hku.hken_US
dc.identifier.authorityCheung, YF=rp00382en_US
dc.description.naturelink_to_subscribed_fulltexten_US
dc.identifier.doi10.1016/S0140-6736(00)02068-7-
dc.identifier.pmid10791382-
dc.identifier.scopuseid_2-s2.0-0034175993en_US
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-0034175993&selection=ref&src=s&origin=recordpageen_US
dc.identifier.volume355en_US
dc.identifier.issue9210en_US
dc.identifier.spage1157en_US
dc.identifier.epage1158en_US
dc.identifier.isiWOS:000086380900017-
dc.publisher.placeUnited Kingdomen_US
dc.identifier.scopusauthoridCheung, YF=7202111067en_US
dc.identifier.scopusauthoridTaylor, MJO=8969782000en_US
dc.identifier.scopusauthoridFisk, NM=7102641855en_US
dc.identifier.scopusauthoridRedington, AN=7102622991en_US
dc.identifier.scopusauthoridGardiner, HM=7004840184en_US
dc.identifier.issnl0140-6736-

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