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Article: BCR-ABL/GATA1/miR-138 mini circuitry contributes to the leukemogenesis of chronic myeloid leukemia

TitleBCR-ABL/GATA1/miR-138 mini circuitry contributes to the leukemogenesis of chronic myeloid leukemia
Authors
KeywordsBCR-ABL
chronic myeloid leukemia
imatinib
microRNA
Issue Date2014
PublisherSpringer Nature [academic journals on nature.com]. The Journal's web site is located at http://www.nature.com/onc
Citation
Oncogene, 2014, v. 33 n. 1, p. 44-54 How to Cite?
DescriptionBronze open access
Persistent Identifierhttp://hdl.handle.net/10722/181994
ISSN
2021 Impact Factor: 8.756
2020 SCImago Journal Rankings: 3.395
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorXu, C-
dc.contributor.authorFu, H-
dc.contributor.authorGao, L-
dc.contributor.authorWang, L-
dc.contributor.authorWang, W-
dc.contributor.authorLi, J-
dc.contributor.authorLi, Y-
dc.contributor.authorDou, L-
dc.contributor.authorGao, X-
dc.contributor.authorLuo, X-
dc.contributor.authorJing, Y-
dc.contributor.authorChim, JCS-
dc.contributor.authorZheng, X-
dc.contributor.authorYu, L-
dc.date.accessioned2013-04-17T07:16:59Z-
dc.date.available2013-04-17T07:16:59Z-
dc.date.issued2014-
dc.identifier.citationOncogene, 2014, v. 33 n. 1, p. 44-54-
dc.identifier.issn0950-9232-
dc.identifier.urihttp://hdl.handle.net/10722/181994-
dc.descriptionBronze open access-
dc.languageeng-
dc.publisherSpringer Nature [academic journals on nature.com]. The Journal's web site is located at http://www.nature.com/onc-
dc.relation.ispartofOncogene-
dc.subjectBCR-ABL-
dc.subjectchronic myeloid leukemia-
dc.subjectimatinib-
dc.subjectmicroRNA-
dc.titleBCR-ABL/GATA1/miR-138 mini circuitry contributes to the leukemogenesis of chronic myeloid leukemia-
dc.typeArticle-
dc.identifier.emailChim, JCS: jcschim@hku.hk-
dc.identifier.authorityChim, JCS=rp00408-
dc.description.naturelink_to_OA_fulltext-
dc.identifier.doi10.1038/onc.2012.557-
dc.identifier.pmid23208504-
dc.identifier.scopuseid_2-s2.0-84891745496-
dc.identifier.hkuros213800-
dc.identifier.volume33-
dc.identifier.issue1-
dc.identifier.spage44-
dc.identifier.epage54-
dc.identifier.isiWOS:000329212000006-
dc.publisher.placeUnited Kingdom-
dc.identifier.issnl0950-9232-

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