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Conference Paper: Adiponectin is protective against oxidative stress-induced cytotoxicity in amyloid-beta neurotoxicity

TitleAdiponectin is protective against oxidative stress-induced cytotoxicity in amyloid-beta neurotoxicity
Authors
Chan, KHLam, KSLCheng, OYKwan, JSCHo, PWLCheng, KKYChung, SKHo, JWMGuo, VYXu, A
KeywordsMedical sciences
Issue Date2013
PublisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org.hk
Citation
The 18th Medical Research Conference (MRC 2013), The University of Hong Kong, Hong Kong, 12 January 2013. In Hong Kong Medical Journal, 2013, v. 19 suppl. 1, p. 10, abstract no. 1 How to Cite?
AbstractBeta-amyloid (Aβ) neurotoxicity is important in Alzheimer’s disease (AD) pathogenesis. Aβ neurotoxicity causes oxidative stress, inflammation, and mitochondrial damage resulting in neuronal degeneration and death. Oxidative stress, inflammation, and mitochondrial failure are also pathophysiological mechanisms of type 2 diabetes mellitus (T2DM) which is characterised by insulin resistance. Interestingly, T2DM increases risk to develop AD which is associated with reduced neuronal insulin sensitivity (central insulin resistance). We studied the potential protective effect of adipon…
DescriptionOral Presentation
Persistent Identifierhttp://hdl.handle.net/10722/182425
ISSN
1024-2708
2023 Impact Factor: 3.1
2023 SCImago Journal Rankings: 0.261

 

DC FieldValueLanguage
dc.contributor.authorChan, KH-
dc.contributor.authorLam, KSL-
dc.contributor.authorCheng, OY-
dc.contributor.authorKwan, JSC-
dc.contributor.authorHo, PWL-
dc.contributor.authorCheng, KKY-
dc.contributor.authorChung, SK-
dc.contributor.authorHo, JWM-
dc.contributor.authorGuo, VY-
dc.contributor.authorXu, A-
dc.date.accessioned2013-04-29T08:12:25Z-
dc.date.available2013-04-29T08:12:25Z-
dc.date.issued2013-
dc.identifier.citationThe 18th Medical Research Conference (MRC 2013), The University of Hong Kong, Hong Kong, 12 January 2013. In Hong Kong Medical Journal, 2013, v. 19 suppl. 1, p. 10, abstract no. 1-
dc.identifier.issn1024-2708-
dc.identifier.urihttp://hdl.handle.net/10722/182425-
dc.descriptionOral Presentation-
dc.description.abstractBeta-amyloid (Aβ) neurotoxicity is important in Alzheimer’s disease (AD) pathogenesis. Aβ neurotoxicity causes oxidative stress, inflammation, and mitochondrial damage resulting in neuronal degeneration and death. Oxidative stress, inflammation, and mitochondrial failure are also pathophysiological mechanisms of type 2 diabetes mellitus (T2DM) which is characterised by insulin resistance. Interestingly, T2DM increases risk to develop AD which is associated with reduced neuronal insulin sensitivity (central insulin resistance). We studied the potential protective effect of adipon…-
dc.languageeng-
dc.publisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org.hk-
dc.relation.ispartofHong Kong Medical Journal-
dc.rightsHong Kong Medical Journal. Copyright © Hong Kong Academy of Medicine Press.-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.subjectMedical sciences-
dc.titleAdiponectin is protective against oxidative stress-induced cytotoxicity in amyloid-beta neurotoxicityen_US
dc.typeConference_Paperen_US
dc.identifier.emailChan, KH: koonho@hku.hk-
dc.identifier.emailLam, KSL: ksllam@hku.hk-
dc.identifier.emailCheng, OY: onyin@hku.hk-
dc.identifier.emailKwan, JSC: jsckwan@hkucc.hku.hk-
dc.identifier.emailHo, PWL: hwl2002@hku.hk-
dc.identifier.emailCheng, KKY: dorncky@hkucc.hku.hk-
dc.identifier.emailChung, SK: skchung@hkucc.hku.hk-
dc.identifier.emailHo, JWM: seeka@hku.hk-
dc.identifier.emailGuo, VY: viviguo@hku.hk-
dc.identifier.emailXu, A: amxu@hkucc.hku.hk-
dc.description.naturepublished_or_final_version-
dc.identifier.hkuros213857-
dc.identifier.volume19-
dc.identifier.issuesuppl. 1-
dc.identifier.spage10, abstract no. 1-
dc.identifier.epage10, abstract no. 1-
dc.publisher.placeHong Kong-
dc.customcontrol.immutablesml 130429-
dc.identifier.issnl1024-2708-

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