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Article: p53 mediates autophagy activation and mitochondria dysfunction in kainic acid-induced excitotoxicity in primary striatal neurons

Titlep53 mediates autophagy activation and mitochondria dysfunction in kainic acid-induced excitotoxicity in primary striatal neurons
Authors
KeywordsAutophagy
Excitotoxicity
Kainic acid
Mitochondria
P53
Issue Date2012
PublisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/neuroscience
Citation
Neuroscience, 2012, v. 207, p. 52-64 How to Cite?
Persistent Identifierhttp://hdl.handle.net/10722/185637
ISSN
2021 Impact Factor: 3.708
2020 SCImago Journal Rankings: 1.297
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorDong, XXen_US
dc.contributor.authorWang, YRen_US
dc.contributor.authorQin, Sen_US
dc.contributor.authorLiang, ZQen_US
dc.contributor.authorLiu, BHen_US
dc.contributor.authorQin, ZHen_US
dc.contributor.authorWang, Yen_US
dc.date.accessioned2013-08-20T11:36:11Z-
dc.date.available2013-08-20T11:36:11Z-
dc.date.issued2012en_US
dc.identifier.citationNeuroscience, 2012, v. 207, p. 52-64en_US
dc.identifier.issn0306-4522en_US
dc.identifier.urihttp://hdl.handle.net/10722/185637-
dc.languageengen_US
dc.publisherElsevier BV. The Journal's web site is located at http://www.elsevier.com/locate/neuroscienceen_US
dc.relation.ispartofNeuroscienceen_US
dc.rightsNOTICE: this is the author’s version of a work that was accepted for publication in <Journal title>. Changes resulting from the publishing process, such as peer review, editing, corrections, structural formatting, and other quality control mechanisms may not be reflected in this document. Changes may have been made to this work since it was submitted for publication. A definitive version was subsequently published in PUBLICATION, [VOL#, ISSUE#, (DATE)] DOI#en_US
dc.subjectAutophagy-
dc.subjectExcitotoxicity-
dc.subjectKainic acid-
dc.subjectMitochondria-
dc.subjectP53-
dc.titlep53 mediates autophagy activation and mitochondria dysfunction in kainic acid-induced excitotoxicity in primary striatal neuronsen_US
dc.typeArticleen_US
dc.identifier.emailLiu, BH: ppliew@hku.hken_US
dc.identifier.authorityLiu, BH=rp01485en_US
dc.identifier.doi10.1016/j.neuroscience.2012.01.018en_US
dc.identifier.pmid22330834-
dc.identifier.scopuseid_2-s2.0-84862826039-
dc.identifier.hkuros216920en_US
dc.identifier.volume207en_US
dc.identifier.spage52en_US
dc.identifier.epage64en_US
dc.identifier.isiWOS:000302203000005-
dc.publisher.placeNetherlandsen_US
dc.identifier.issnl0306-4522-

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