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Article: Repression of integrin-linked kinase by antidiabetes drugs through cross-talk of PPARγ- and AMPKα-dependent signaling: role of AP-2α and Sp1

TitleRepression of integrin-linked kinase by antidiabetes drugs through cross-talk of PPARγ- and AMPKα-dependent signaling: role of AP-2α and Sp1
Authors
Hahn, SSTang, QZheng, FZhao, SWu, JChen, J
KeywordsAMPKα
AP-2α
ILK
Nasopharyngeal carcinoma cells
PPARγ
Sp1
Issue Date2014
PublisherElsevier Inc. The Journal's web site is located at http://www.elsevier.com/locate/cellsig
Citation
Cellular Signalling, 2014, v. 26 n. 3, p. 639-647 How to Cite?
DOI: http://dx.doi.org/10.1016/j.cellsig.2013.12.004
Persistent Identifierhttp://hdl.handle.net/10722/196260
ISSN
0898-6568
2023 Impact Factor: 4.4
2023 SCImago Journal Rankings: 1.317
ISI Accession Number ID
WOS:000331159700020

 

DC FieldValueLanguage
dc.contributor.authorHahn, SS-
dc.contributor.authorTang, Q-
dc.contributor.authorZheng, F-
dc.contributor.authorZhao, S-
dc.contributor.authorWu, J-
dc.contributor.authorChen, J-
dc.date.accessioned2014-04-01T04:05:27Z-
dc.date.available2014-04-01T04:05:27Z-
dc.date.issued2014-
dc.identifier.citationCellular Signalling, 2014, v. 26 n. 3, p. 639-647-
dc.identifier.issn0898-6568-
dc.identifier.urihttp://hdl.handle.net/10722/196260-
dc.languageeng-
dc.publisherElsevier Inc. The Journal's web site is located at http://www.elsevier.com/locate/cellsig-
dc.relation.ispartofCellular Signalling-
dc.subjectAMPKα-
dc.subjectAP-2α-
dc.subjectILK-
dc.subjectNasopharyngeal carcinoma cells-
dc.subjectPPARγ-
dc.subjectSp1-
dc.titleRepression of integrin-linked kinase by antidiabetes drugs through cross-talk of PPARγ- and AMPKα-dependent signaling: role of AP-2α and Sp1en_US
dc.typeArticleen_US
dc.identifier.emailChen, J: abchen@hku.hk-
dc.identifier.doi10.1016/j.cellsig.2013.12.004-
dc.identifier.pmid24361375-
dc.identifier.scopuseid_2-s2.0-84891808975-
dc.identifier.hkuros228358-
dc.identifier.volume26-
dc.identifier.issue3-
dc.identifier.spage639-
dc.identifier.epage647-
dc.identifier.isiWOS:000331159700020-
dc.publisher.placeUnited States-
dc.identifier.issnl0898-6568-

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