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Article: Selective inhibition of PTEN preserves ischaemic post-conditioning cardioprotection in STZ-induced Type 1 diabetic rats: role of the PI3K/Akt and JAK2/STAT3 pathways

TitleSelective inhibition of PTEN preserves ischaemic post-conditioning cardioprotection in STZ-induced Type 1 diabetic rats: role of the PI3K/Akt and JAK2/STAT3 pathways
Authors
KeywordsDiabetes
Ischaemic post-conditioning
Myocardial ischaemia/reperfusion injury
Phosphatase and tensin homologue deleted on chromosome 10 (PTEN)
Issue Date2016
Citation
Clinical Science, 2016, v. 130, p. 377-392 How to Cite?
Persistent Identifierhttp://hdl.handle.net/10722/234018
ISSN
2023 Impact Factor: 6.7
2023 SCImago Journal Rankings: 1.565
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorXue, R-
dc.contributor.authorLei, S-
dc.contributor.authorXia, Z-
dc.contributor.authorWu, Y-
dc.contributor.authorMeng, Q-
dc.contributor.authorZhan, L-
dc.contributor.authorSu, W-
dc.contributor.authorLiu, H-
dc.contributor.authorXu, J-
dc.contributor.authorLiu, Z-
dc.contributor.authorZhou, B-
dc.contributor.authorXia, Z-
dc.date.accessioned2016-10-14T06:58:30Z-
dc.date.available2016-10-14T06:58:30Z-
dc.date.issued2016-
dc.identifier.citationClinical Science, 2016, v. 130, p. 377-392-
dc.identifier.issn0143-5221-
dc.identifier.urihttp://hdl.handle.net/10722/234018-
dc.languageeng-
dc.relation.ispartofClinical Science-
dc.subjectDiabetes-
dc.subjectIschaemic post-conditioning-
dc.subjectMyocardial ischaemia/reperfusion injury-
dc.subjectPhosphatase and tensin homologue deleted on chromosome 10 (PTEN)-
dc.titleSelective inhibition of PTEN preserves ischaemic post-conditioning cardioprotection in STZ-induced Type 1 diabetic rats: role of the PI3K/Akt and JAK2/STAT3 pathways-
dc.typeArticle-
dc.identifier.emailXia, Z: zyxia@hkucc.hku.hk-
dc.identifier.authorityXia, Z=rp00532-
dc.identifier.doi10.1042/CS20150496-
dc.identifier.scopuseid_2-s2.0-84958092745-
dc.identifier.hkuros267541-
dc.identifier.volume130-
dc.identifier.spage377-
dc.identifier.epage392-
dc.identifier.eissn1470-8736-
dc.identifier.isiWOS:000369718400006-
dc.identifier.issnl0143-5221-

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