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Article: Alteration of the immune cell profiles in the pathophysiology of tubal ectopic pregnancy

TitleAlteration of the immune cell profiles in the pathophysiology of tubal ectopic pregnancy
Authors
Wang, XLee, CLLi, RHWVijayan, MDuan, YYeung, WSBZhang, YChiu, PCN
Keywordsfallopian tube
macrophages
natural killer cell
T cell
tubal ectopic pregnancy
Issue Date2019
PublisherWiley-Blackwell Publishing, Inc. The Journal's web site is located at http://onlinelibrary.wiley.com/journal/10.1111/(ISSN)1600-0897
Citation
American Journal of Reproductive Immunology, 2019, v. 81 n. 4, p. article no. e13093 How to Cite?
DOI: http://dx.doi.org/10.1111/aji.13093
AbstractTubal ectopic pregnancy (TEP) refers to implantation of conceptus in the fallopian tube. It makes up over 98% of ectopic pregnancy (EP), which is the leading cause of maternal morbidity and mortality in the first trimester of pregnancy. Immune cells at the maternal‐fetal interface play important roles in the process of embryo implantation, stroma decidualization, and early placental development. Alterations in the composition, phenotype, and activity of the immune cells in the fallopian tubes contribute toward the onset of TEP. In this review, we compare the leukocytic proportions in decidua of normal pregnancy, and in decidua and fallopian tubes of TEP. The possible functions of these immune cells in the pathophysiology of TEP are also discussed.
Persistent Identifierhttp://hdl.handle.net/10722/274097
ISSN
1046-7408
2023 Impact Factor: 2.5
2023 SCImago Journal Rankings: 0.887
ISI Accession Number ID
WOS:000466389900006

 

DC FieldValueLanguage
dc.contributor.authorWang, X-
dc.contributor.authorLee, CL-
dc.contributor.authorLi, RHW-
dc.contributor.authorVijayan, M-
dc.contributor.authorDuan, Y-
dc.contributor.authorYeung, WSB-
dc.contributor.authorZhang, Y-
dc.contributor.authorChiu, PCN-
dc.date.accessioned2019-08-18T14:55:00Z-
dc.date.available2019-08-18T14:55:00Z-
dc.date.issued2019-
dc.identifier.citationAmerican Journal of Reproductive Immunology, 2019, v. 81 n. 4, p. article no. e13093-
dc.identifier.issn1046-7408-
dc.identifier.urihttp://hdl.handle.net/10722/274097-
dc.description.abstractTubal ectopic pregnancy (TEP) refers to implantation of conceptus in the fallopian tube. It makes up over 98% of ectopic pregnancy (EP), which is the leading cause of maternal morbidity and mortality in the first trimester of pregnancy. Immune cells at the maternal‐fetal interface play important roles in the process of embryo implantation, stroma decidualization, and early placental development. Alterations in the composition, phenotype, and activity of the immune cells in the fallopian tubes contribute toward the onset of TEP. In this review, we compare the leukocytic proportions in decidua of normal pregnancy, and in decidua and fallopian tubes of TEP. The possible functions of these immune cells in the pathophysiology of TEP are also discussed.-
dc.languageeng-
dc.publisherWiley-Blackwell Publishing, Inc. The Journal's web site is located at http://onlinelibrary.wiley.com/journal/10.1111/(ISSN)1600-0897-
dc.relation.ispartofAmerican Journal of Reproductive Immunology-
dc.rightsPreprint This is the pre-peer reviewed version of the following article: [FULL CITE], which has been published in final form at [Link to final article using the DOI]. This article may be used for non-commercial purposes in accordance with Wiley Terms and Conditions for Use of Self-Archived Versions. Postprint This is the peer reviewed version of the following article: [FULL CITE], which has been published in final form at [Link to final article using the DOI]. This article may be used for non-commercial purposes in accordance with Wiley Terms and Conditions for Use of Self-Archived Versions.-
dc.subjectfallopian tube-
dc.subjectmacrophages-
dc.subjectnatural killer cell-
dc.subjectT cell-
dc.subjecttubal ectopic pregnancy-
dc.titleAlteration of the immune cell profiles in the pathophysiology of tubal ectopic pregnancy-
dc.typeArticle-
dc.identifier.emailLee, CL: kcllee@hku.hk-
dc.identifier.emailLi, RHW: raymondli@hku.hk-
dc.identifier.emailYeung, WSB: wsbyeung@hku.hk-
dc.identifier.emailChiu, PCN: pchiucn@hku.hk-
dc.identifier.authorityLee, CL=rp02515-
dc.identifier.authorityLi, RHW=rp01649-
dc.identifier.authorityYeung, WSB=rp00331-
dc.identifier.authorityChiu, PCN=rp00424-
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1111/aji.13093-
dc.identifier.pmid30672642-
dc.identifier.scopuseid_2-s2.0-85061768610-
dc.identifier.hkuros302138-
dc.identifier.volume81-
dc.identifier.issue4-
dc.identifier.spagearticle no. e13093-
dc.identifier.epagearticle no. e13093-
dc.identifier.isiWOS:000466389900006-
dc.publisher.placeUnited States-
dc.identifier.issnl1046-7408-

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