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Article: Antiviral activity of human γδ-T cells against enterovirus 71

TitleAntiviral activity of human γδ-T cells against enterovirus 71
Authors
Issue Date2019
PublisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org/
Citation
Hong Kong Medical Journal, 2019, v. 25 n. 6, suppl. 9, p. 21-23 How to Cite?
Abstract1. Vγ9Vδ2-T cells can directly kill enterovirus (EV) 71–infected cells. 2. The cytotoxicity of Vγ9Vδ2-T cells against EV71-infected cells is mediated by the Fas-FasL pathway. 3. Vγ9Vδ2-T cells have non-cytolytic antiviral activity against EV71 through their secreted IFN-γ.
Persistent Identifierhttp://hdl.handle.net/10722/289279
ISSN
2023 Impact Factor: 3.1
2023 SCImago Journal Rankings: 0.261

 

DC FieldValueLanguage
dc.contributor.authorLam, KT-
dc.contributor.authorZheng, J-
dc.contributor.authorXiang, Z-
dc.contributor.authorLiu, YP-
dc.contributor.authorLau, YL-
dc.contributor.authorTu, WW-
dc.date.accessioned2020-10-22T08:10:24Z-
dc.date.available2020-10-22T08:10:24Z-
dc.date.issued2019-
dc.identifier.citationHong Kong Medical Journal, 2019, v. 25 n. 6, suppl. 9, p. 21-23-
dc.identifier.issn1024-2708-
dc.identifier.urihttp://hdl.handle.net/10722/289279-
dc.description.abstract1. Vγ9Vδ2-T cells can directly kill enterovirus (EV) 71–infected cells. 2. The cytotoxicity of Vγ9Vδ2-T cells against EV71-infected cells is mediated by the Fas-FasL pathway. 3. Vγ9Vδ2-T cells have non-cytolytic antiviral activity against EV71 through their secreted IFN-γ.-
dc.languageeng-
dc.publisherHong Kong Academy of Medicine Press. The Journal's web site is located at http://www.hkmj.org/-
dc.relation.ispartofHong Kong Medical Journal-
dc.titleAntiviral activity of human γδ-T cells against enterovirus 71-
dc.typeArticle-
dc.identifier.emailLam, KT: tailam@hkucc.hku.hk-
dc.identifier.emailLiu, YP: yinpingl@hku.hk-
dc.identifier.emailLau, YL: lauylung@hku.hk-
dc.identifier.emailTu, WW: wwtu@hku.hk-
dc.identifier.authorityZheng, J=rp02004-
dc.identifier.authorityLiu, YP=rp00269-
dc.identifier.authorityLau, YL=rp00361-
dc.identifier.authorityTu, WW=rp00416-
dc.description.naturelink_to_OA_fulltext-
dc.identifier.pmid31889030-
dc.identifier.scopuseid_2-s2.0-85077307463-
dc.identifier.hkuros316635-
dc.identifier.volume25-
dc.identifier.issue6, suppl. 9-
dc.identifier.spage21-
dc.identifier.epage23-
dc.publisher.placeHong Kong-

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