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Article: Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes

TitleCancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes
Authors
Issue Date2020
PublisherNature Research: Fully open access journals. The Journal's web site is located at http://www.nature.com/sigtrans/
Citation
Signal Transduction and Targeted Therapy, 2020, v. 5 n. 1, p. article no. 27 How to Cite?
Persistent Identifierhttp://hdl.handle.net/10722/293495
ISSN
2020 SCImago Journal Rankings: 4.284
PubMed Central ID
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorWan, YCE-
dc.contributor.authorLeung, TCS-
dc.contributor.authorDing, D-
dc.contributor.authorSun, X-
dc.contributor.authorLiu, J-
dc.contributor.authorZhu, L-
dc.contributor.authorKang, TZE-
dc.contributor.authorYang, D-
dc.contributor.authorZhang, Y-
dc.contributor.authorZhang, J-
dc.contributor.authorQian, C-
dc.contributor.authorHuen, MSY-
dc.contributor.authorLi, Q-
dc.contributor.authorChow, MZY-
dc.contributor.authorZheng, Z-
dc.contributor.authorHan, J-
dc.contributor.authorGoel, A-
dc.contributor.authorWang, X-
dc.contributor.authorIshibashi, T-
dc.contributor.authorChan, KM-
dc.date.accessioned2020-11-23T08:17:36Z-
dc.date.available2020-11-23T08:17:36Z-
dc.date.issued2020-
dc.identifier.citationSignal Transduction and Targeted Therapy, 2020, v. 5 n. 1, p. article no. 27-
dc.identifier.issn2059-3635-
dc.identifier.urihttp://hdl.handle.net/10722/293495-
dc.languageeng-
dc.publisherNature Research: Fully open access journals. The Journal's web site is located at http://www.nature.com/sigtrans/-
dc.relation.ispartofSignal Transduction and Targeted Therapy-
dc.rightsSignal Transduction and Targeted Therapy. Copyright © Nature Research: Fully open access journals.-
dc.rightsThis work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License.-
dc.titleCancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes-
dc.typeArticle-
dc.identifier.emailQian, C: cmqian@hku.hk-
dc.identifier.emailHuen, MSY: huen.michael@hku.hk-
dc.identifier.authorityQian, C=rp01371-
dc.identifier.authorityHuen, MSY=rp01336-
dc.description.naturepublished_or_final_version-
dc.identifier.doi10.1038/s41392-020-0131-0-
dc.identifier.pmid32296031-
dc.identifier.pmcidPMC7060176-
dc.identifier.scopuseid_2-s2.0-85082020260-
dc.identifier.hkuros319293-
dc.identifier.volume5-
dc.identifier.issue1-
dc.identifier.spagearticle no. 27-
dc.identifier.epagearticle no. 27-
dc.identifier.isiWOS:000518366800001-
dc.publisher.placeUnited States-

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