File Download
Supplementary
-
Citations:
- Appears in Collections:
Conference Paper: Anti-beta2 glycoprotein I (beta2GPI) antibodies facilitates phagocytosis of apoptotic neutrophils in patients with systemic lupus erythematosus (SLE)
Title | Anti-beta2 glycoprotein I (beta2GPI) antibodies facilitates phagocytosis of apoptotic neutrophils in patients with systemic lupus erythematosus (SLE) |
---|---|
Authors | |
Issue Date | 2003 |
Publisher | Hong Kong Academy of Medicine Press. The Journal's website is located at http://www.hkmj.org/resources/supp.html |
Citation | The 8th Medical Research Conference (MRC 2003), Medical Science Group, Queen Mary Hospital, Hong Kong, 25-26 January 2003. In Hong Kong Medical Journal, 2003, v. 9 n. 1 suppl., p. 65, abstract no. NRI-01 How to Cite? |
Abstract | BACKGROUND : Increased apoptosis provides a continuous source autoantigens and has been suggested to have a pathogenic role in SLE. We have previously shown patients with SLE have impaired clearance of apoptotic bodies. In this study, we evaluated whether anti-beta2GPI antibodies, a subset of antiphospholipid antibodies, may have an effect on the phagocytosis of apoptotic bodies by macrophages in these patients … |
Persistent Identifier | http://hdl.handle.net/10722/54128 |
ISSN | 2023 Impact Factor: 3.1 2023 SCImago Journal Rankings: 0.261 |
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Mok, TMY | en_HK |
dc.contributor.author | Yeung, JSL | en_HK |
dc.contributor.author | Jin, O | en_HK |
dc.contributor.author | Chan, WK | en_HK |
dc.contributor.author | Lo, Y | en_HK |
dc.contributor.author | Lau, WCS | en_HK |
dc.date.accessioned | 2009-04-03T07:37:30Z | - |
dc.date.available | 2009-04-03T07:37:30Z | - |
dc.date.issued | 2003 | en_HK |
dc.identifier.citation | The 8th Medical Research Conference (MRC 2003), Medical Science Group, Queen Mary Hospital, Hong Kong, 25-26 January 2003. In Hong Kong Medical Journal, 2003, v. 9 n. 1 suppl., p. 65, abstract no. NRI-01 | en_HK |
dc.identifier.issn | 1024-2708 | en_HK |
dc.identifier.uri | http://hdl.handle.net/10722/54128 | - |
dc.description.abstract | BACKGROUND : Increased apoptosis provides a continuous source autoantigens and has been suggested to have a pathogenic role in SLE. We have previously shown patients with SLE have impaired clearance of apoptotic bodies. In this study, we evaluated whether anti-beta2GPI antibodies, a subset of antiphospholipid antibodies, may have an effect on the phagocytosis of apoptotic bodies by macrophages in these patients … | - |
dc.language | eng | en_HK |
dc.publisher | Hong Kong Academy of Medicine Press. The Journal's website is located at http://www.hkmj.org/resources/supp.html | en_HK |
dc.relation.ispartof | Hong Kong Medical Journal | - |
dc.rights | Hong Kong Medical Journal. Copyright © Hong Kong Academy of Medicine Press. | en_HK |
dc.rights | This work is licensed under a Creative Commons Attribution-NonCommercial-NoDerivatives 4.0 International License. | - |
dc.title | Anti-beta2 glycoprotein I (beta2GPI) antibodies facilitates phagocytosis of apoptotic neutrophils in patients with systemic lupus erythematosus (SLE) | en_HK |
dc.type | Conference_Paper | en_HK |
dc.identifier.openurl | http://library.hku.hk:4550/resserv?sid=HKU:IR&issn=1024-2708&volume=9&issue=1 Suppl&spage=65&epage=&date=2003&atitle=Anti-beta2+glycoprotein+I+(beta2GPI)+antibodies+facilitates+phagocytosis+of+apoptotic+neutrophils+in+patients+with+systemic+lupus+erythematosus+(SLE) | en_HK |
dc.identifier.email | Mok, TMY: temy@hkucc.hku.hk | en_HK |
dc.identifier.email | Yeung, JSL: joebecky@hotmail.com | en_HK |
dc.identifier.email | Chan, WK: wkchanf@HKUSUA.hku.hk | en_HK |
dc.identifier.email | Lo, Y: yloa@hkucc.hku.hk | en_HK |
dc.identifier.email | Lau, WCS: cslau@hku.hk | en_HK |
dc.description.nature | published_or_final_version | en_HK |
dc.identifier.hkuros | 95861 | - |
dc.identifier.hkuros | 85435 | - |
dc.identifier.volume | 9 | - |
dc.identifier.issue | 1 suppl. | - |
dc.identifier.spage | 65, abstract no. NRI-01 | - |
dc.identifier.epage | 65, abstract no. NRI-01 | - |
dc.identifier.issnl | 1024-2708 | - |