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Article: Role of domestic ducks in the propagation and biological evolution of highly pathogenic H5N1 influenza viruses in Asia

TitleRole of domestic ducks in the propagation and biological evolution of highly pathogenic H5N1 influenza viruses in Asia
Authors
HulsePost, DJSturmRamirez, KMHumberd, JSeiler, PGovorkova, EAKrauss, SScholtissek, CPuthavathana, PBuranathai, CNguyen, TDLong, HTNaipospos, TSPChen, HEllis, TMGuan, YPeiris, JSMWebster, RG
KeywordsAvian influenza
Pathogenicity
Issue Date2005
PublisherNational Academy of Sciences. The Journal's web site is located at http://www.pnas.org
Citation
Proceedings Of The National Academy Of Sciences Of The United States Of America, 2005, v. 102 n. 30, p. 10682-10687 How to Cite?
DOI: http://dx.doi.org/10.1073/pnas.0504662102
AbstractWild waterfowl, including ducks, are natural hosts of influenza A viruses. These viruses rarely caused disease in ducks until 2002, when some H5N1 strains became highly pathogenic. Here we show that these H5N1 viruses are reverting to nonpathogenicity in ducks. Ducks experimentally infected with viruses isolated between 2003 and 2004 shed virus for an extended time (up to 17 days), during which variant viruses with low pathogenicity were selected. These results suggest that the duck has become the "Trojan horse" of Asian H5N1 influenza viruses. The ducks that are unaffected by infection with these viruses continue to circulate these viruses, presenting a pandemic threat. © 2005 by The National Academy of Sciences of the USA.
Persistent Identifierhttp://hdl.handle.net/10722/78922
ISSN
0027-8424
2022 Impact Factor: 11.1
2020 SCImago Journal Rankings: 5.011
ISI Accession Number ID
WOS:000230853300050
References
References in Scopus

 

DC FieldValueLanguage
dc.contributor.authorHulsePost, DJen_HK
dc.contributor.authorSturmRamirez, KMen_HK
dc.contributor.authorHumberd, Jen_HK
dc.contributor.authorSeiler, Pen_HK
dc.contributor.authorGovorkova, EAen_HK
dc.contributor.authorKrauss, Sen_HK
dc.contributor.authorScholtissek, Cen_HK
dc.contributor.authorPuthavathana, Pen_HK
dc.contributor.authorBuranathai, Cen_HK
dc.contributor.authorNguyen, TDen_HK
dc.contributor.authorLong, HTen_HK
dc.contributor.authorNaipospos, TSPen_HK
dc.contributor.authorChen, Hen_HK
dc.contributor.authorEllis, TMen_HK
dc.contributor.authorGuan, Yen_HK
dc.contributor.authorPeiris, JSMen_HK
dc.contributor.authorWebster, RGen_HK
dc.date.accessioned2010-09-06T07:48:26Z-
dc.date.available2010-09-06T07:48:26Z-
dc.date.issued2005en_HK
dc.identifier.citationProceedings Of The National Academy Of Sciences Of The United States Of America, 2005, v. 102 n. 30, p. 10682-10687en_HK
dc.identifier.issn0027-8424en_HK
dc.identifier.urihttp://hdl.handle.net/10722/78922-
dc.description.abstractWild waterfowl, including ducks, are natural hosts of influenza A viruses. These viruses rarely caused disease in ducks until 2002, when some H5N1 strains became highly pathogenic. Here we show that these H5N1 viruses are reverting to nonpathogenicity in ducks. Ducks experimentally infected with viruses isolated between 2003 and 2004 shed virus for an extended time (up to 17 days), during which variant viruses with low pathogenicity were selected. These results suggest that the duck has become the "Trojan horse" of Asian H5N1 influenza viruses. The ducks that are unaffected by infection with these viruses continue to circulate these viruses, presenting a pandemic threat. © 2005 by The National Academy of Sciences of the USA.en_HK
dc.languageengen_HK
dc.publisherNational Academy of Sciences. The Journal's web site is located at http://www.pnas.orgen_HK
dc.relation.ispartofProceedings of the National Academy of Sciences of the United States of Americaen_HK
dc.subjectAvian influenzaen_HK
dc.subjectPathogenicityen_HK
dc.titleRole of domestic ducks in the propagation and biological evolution of highly pathogenic H5N1 influenza viruses in Asiaen_HK
dc.typeArticleen_HK
dc.identifier.emailGuan, Y: yguan@hkucc.hku.hken_HK
dc.identifier.emailPeiris, JSM: malik@hkucc.hku.hken_HK
dc.identifier.authorityGuan, Y=rp00397en_HK
dc.identifier.authorityPeiris, JSM=rp00410en_HK
dc.description.naturelink_to_subscribed_fulltext-
dc.identifier.doi10.1073/pnas.0504662102en_HK
dc.identifier.pmid16030144-
dc.identifier.scopuseid_2-s2.0-23044502533en_HK
dc.identifier.hkuros110177en_HK
dc.relation.referenceshttp://www.scopus.com/mlt/select.url?eid=2-s2.0-23044502533&selection=ref&src=s&origin=recordpageen_HK
dc.identifier.volume102en_HK
dc.identifier.issue30en_HK
dc.identifier.spage10682en_HK
dc.identifier.epage10687en_HK
dc.identifier.isiWOS:000230853300050-
dc.publisher.placeUnited Statesen_HK
dc.identifier.scopusauthoridHulsePost, DJ=11940084300en_HK
dc.identifier.scopusauthoridSturmRamirez, KM=6507645950en_HK
dc.identifier.scopusauthoridHumberd, J=6506769380en_HK
dc.identifier.scopusauthoridSeiler, P=7005281020en_HK
dc.identifier.scopusauthoridGovorkova, EA=7003837718en_HK
dc.identifier.scopusauthoridKrauss, S=7102769210en_HK
dc.identifier.scopusauthoridScholtissek, C=7006002772en_HK
dc.identifier.scopusauthoridPuthavathana, P=7004642707en_HK
dc.identifier.scopusauthoridBuranathai, C=6507522489en_HK
dc.identifier.scopusauthoridNguyen, TD=25227791100en_HK
dc.identifier.scopusauthoridLong, HT=7102434129en_HK
dc.identifier.scopusauthoridNaipospos, TSP=8722763000en_HK
dc.identifier.scopusauthoridChen, H=8934249400en_HK
dc.identifier.scopusauthoridEllis, TM=7202589926en_HK
dc.identifier.scopusauthoridGuan, Y=7202924055en_HK
dc.identifier.scopusauthoridPeiris, JSM=7005486823en_HK
dc.identifier.scopusauthoridWebster, RG=36048363100en_HK
dc.identifier.issnl0027-8424-

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