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Article: Cancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes

TitleCancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes
Authors
Wan, YCELeung, TCSDing, DSun, XLiu, JZhu, LKan, TZEYang, DZhang, YZhang, JQian, CHuen, MSYLi, QChow, MZYZheng, ZHan, JGoel, AWang, XIshibashi, TChan, KM
Issue Date2020
Citation
Signal Transduction and Targeted Therapy, 2020, v. 5 How to Cite?
DOI: http://dx.doi.org/10.1038/s41392-020-0131-0
Persistent Identifierhttp://hdl.handle.net/10722/293495

 

DC FieldValueLanguage
dc.contributor.authorWan, YCE-
dc.contributor.authorLeung, TCS-
dc.contributor.authorDing, D-
dc.contributor.authorSun, X-
dc.contributor.authorLiu, J-
dc.contributor.authorZhu, L-
dc.contributor.authorKan, TZE-
dc.contributor.authorYang, D-
dc.contributor.authorZhang, Y-
dc.contributor.authorZhang, J-
dc.contributor.authorQian, C-
dc.contributor.authorHuen, MSY-
dc.contributor.authorLi, Q-
dc.contributor.authorChow, MZY-
dc.contributor.authorZheng, Z-
dc.contributor.authorHan, J-
dc.contributor.authorGoel, A-
dc.contributor.authorWang, X-
dc.contributor.authorIshibashi, T-
dc.contributor.authorChan, KM-
dc.date.accessioned2020-11-23T08:17:36Z-
dc.date.available2020-11-23T08:17:36Z-
dc.date.issued2020-
dc.identifier.citationSignal Transduction and Targeted Therapy, 2020, v. 5-
dc.identifier.urihttp://hdl.handle.net/10722/293495-
dc.languageeng-
dc.relation.ispartofSignal Transduction and Targeted Therapy-
dc.titleCancer-associated histone mutation H2BG53D disrupts DNA–histone octamer interaction and promotes oncogenic phenotypes-
dc.typeArticle-
dc.identifier.emailQian, C: cmqian@hku.hk-
dc.identifier.emailHuen, MSY: huen.michael@hku.hk-
dc.identifier.authorityQian, C=rp01371-
dc.identifier.authorityHuen, MSY=rp01336-
dc.identifier.doi10.1038/s41392-020-0131-0-
dc.identifier.hkuros319293-
dc.identifier.volume5-

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