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Conference Paper: Inhibition of both physiological and aberrant Wnt/β-catenin signaling activities associated with stemness in nasopharyngeal carcinoma
Title | Inhibition of both physiological and aberrant Wnt/β-catenin signaling activities associated with stemness in nasopharyngeal carcinoma |
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Other Titles | Inhibition of both physiological and aberrant Wnt/b-catenin signaling activities associated with stemness in NPC |
Authors | |
Keywords | Medical sciences Oncology |
Issue Date | 2014 |
Publisher | American Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/ |
Citation | The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1938 How to Cite? |
Abstract | We previously demonstrated that basic or physiological levels of β-catenin signaling and tissue context play decisional roles in the regulation of self-renewal networks in nasopharyngeal carcinoma (NPC) HONE1 cells. Introduction of physiological levels of β-catenin signaling in HONE1 hybrid cells plays a central role in the control of other pathway activities, such as TGF-β, Activin, and pluripotency maintenance (LIFR and IL6ST), during the stemness transition process. These results revealed novel regulatory ... |
Description | Tumor Biology - Poster Presentations - Proffered Abstracts - Poster Presentations - Developmental Pathways in Cancer: abstract no. 1938 This journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014 ... |
Persistent Identifier | http://hdl.handle.net/10722/198636 |
ISSN | 2023 Impact Factor: 12.5 2023 SCImago Journal Rankings: 3.468 |
ISI Accession Number ID |
DC Field | Value | Language |
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dc.contributor.author | Cheng, Y | - |
dc.contributor.author | Phoon, YP | - |
dc.contributor.author | Lung, ML | - |
dc.date.accessioned | 2014-07-07T08:26:46Z | - |
dc.date.available | 2014-07-07T08:26:46Z | - |
dc.date.issued | 2014 | - |
dc.identifier.citation | The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 1938 | - |
dc.identifier.issn | 0008-5472 | - |
dc.identifier.uri | http://hdl.handle.net/10722/198636 | - |
dc.description | Tumor Biology - Poster Presentations - Proffered Abstracts - Poster Presentations - Developmental Pathways in Cancer: abstract no. 1938 | - |
dc.description | This journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014 ... | - |
dc.description.abstract | We previously demonstrated that basic or physiological levels of β-catenin signaling and tissue context play decisional roles in the regulation of self-renewal networks in nasopharyngeal carcinoma (NPC) HONE1 cells. Introduction of physiological levels of β-catenin signaling in HONE1 hybrid cells plays a central role in the control of other pathway activities, such as TGF-β, Activin, and pluripotency maintenance (LIFR and IL6ST), during the stemness transition process. These results revealed novel regulatory ... | - |
dc.language | eng | - |
dc.publisher | American Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/ | - |
dc.relation.ispartof | Cancer Research | - |
dc.subject | Medical sciences | - |
dc.subject | Oncology | - |
dc.title | Inhibition of both physiological and aberrant Wnt/β-catenin signaling activities associated with stemness in nasopharyngeal carcinoma | - |
dc.title.alternative | Inhibition of both physiological and aberrant Wnt/b-catenin signaling activities associated with stemness in NPC | - |
dc.type | Conference_Paper | - |
dc.identifier.email | Cheng, Y: yuecheng@hku.hk | - |
dc.identifier.email | Phoon, YP: yeepeng@hku.hk | - |
dc.identifier.email | Lung, ML: mlilung@hku.hk | - |
dc.identifier.authority | Cheng, Y=rp01320 | - |
dc.identifier.authority | Lung, ML=rp00300 | - |
dc.description.nature | postprint | - |
dc.identifier.doi | 10.1158/1538-7445.AM2014-1938 | - |
dc.identifier.hkuros | 229924 | - |
dc.identifier.hkuros | 240415 | - |
dc.identifier.volume | 74 | - |
dc.identifier.issue | 19 suppl., abstract no. 1938 | - |
dc.identifier.isi | WOS:000349906902153 | - |
dc.publisher.place | United States | - |
dc.identifier.issnl | 0008-5472 | - |