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Conference Paper: Liver regeneration was attenuated by deletion of CXCL10 or its receptor CXCR3 via YAP1 signaling

TitleLiver regeneration was attenuated by deletion of CXCL10 or its receptor CXCR3 via YAP1 signaling
Authors
Issue Date2016
PublisherLippincott Williams & Wilkins. The Journal's web site is located at http://www.transplantjournal.com
Citation
The 26th International Congress of The Transplantation Society, Hong Kong, China,18-23 August 2016. In Transplantation, 2016, v. 100 n. Suppl. 7S-1, p. S67, abstract no. 328.6 How to Cite?
DescriptionOral Presentation
Persistent Identifierhttp://hdl.handle.net/10722/246172
ISSN
2023 Impact Factor: 5.3
2023 SCImago Journal Rankings: 1.371

 

DC FieldValueLanguage
dc.contributor.authorMa, YY-
dc.contributor.authorLi, C-
dc.contributor.authorGeng, W-
dc.contributor.authorQi, X-
dc.contributor.authorNg, KTP-
dc.contributor.authorMan, K-
dc.contributor.authorLo, CM-
dc.date.accessioned2017-09-18T02:23:42Z-
dc.date.available2017-09-18T02:23:42Z-
dc.date.issued2016-
dc.identifier.citationThe 26th International Congress of The Transplantation Society, Hong Kong, China,18-23 August 2016. In Transplantation, 2016, v. 100 n. Suppl. 7S-1, p. S67, abstract no. 328.6-
dc.identifier.issn0041-1337-
dc.identifier.urihttp://hdl.handle.net/10722/246172-
dc.descriptionOral Presentation-
dc.languageeng-
dc.publisherLippincott Williams & Wilkins. The Journal's web site is located at http://www.transplantjournal.com-
dc.relation.ispartofTransplantation-
dc.titleLiver regeneration was attenuated by deletion of CXCL10 or its receptor CXCR3 via YAP1 signaling-
dc.typeConference_Paper-
dc.identifier.emailQi, X: qixiang515@connect.hku.hk-
dc.identifier.emailNg, KTP: ledodes@hku.hk-
dc.identifier.emailMan, K: kwanman@hku.hk-
dc.identifier.emailLo, CM: chungmlo@hkucc.hku.hk-
dc.identifier.authorityNg, KTP=rp01720-
dc.identifier.authorityMan, K=rp00417-
dc.identifier.authorityLo, CM=rp00412-
dc.identifier.doi10.1097/01.tp.0000490147.72544.1a-
dc.identifier.hkuros278159-
dc.identifier.volume100-
dc.identifier.issueSuppl. 7S-1-
dc.identifier.spageS67, abstract no. 328.6-
dc.identifier.epageS67, abstract no. 328.6-
dc.publisher.placeUnited States-
dc.customcontrol.immutablejt 2017-11-22-
dc.identifier.issnl0041-1337-

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