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Conference Paper: Adverse effects of TERT-CLPTM1L and double-strand breaks repair contribute to risk for nasopharyngeal carcinoma

TitleAdverse effects of TERT-CLPTM1L and double-strand breaks repair contribute to risk for nasopharyngeal carcinoma
Other TitlesAdverse effects if TERT-CLPTM1L and double-strand breaks repair contribute to risk for NPC
Authors
KeywordsMedical sciences
Oncology
Issue Date2014
PublisherAmerican Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/
Citation
The 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 4148 How to Cite?
AbstractBACKGROUND AND AIMS: The genetic etiology of NPC and mechanisms for inherited susceptibility remain unclear. Only modest low-penetrance effects of cancer-predisposing common variant SNPs were previously identified in the few large-scale NPC association studies reported. Most NPC association studies focused on single or limited candidate genes with modest sample sizes. Systematic and comprehensive study designs for evaluation of higher order gene-gene interactions are scanty. A large-scale NPC case-control SNP association study was performed to examine the genetic risk factors for NPC development. In order to elucidate the ...
DescriptionEpidemiology - Poster Presentations - Proffered Abstracts - Poster Presentations - Molecular and Genetic Epidemiology of Lung, Head and Neck, and Gastrointestinal Cancers: abstract no. 4148
This journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA
Persistent Identifierhttp://hdl.handle.net/10722/198639
ISSN
2021 Impact Factor: 13.312
2020 SCImago Journal Rankings: 4.103
ISI Accession Number ID

 

DC FieldValueLanguage
dc.contributor.authorLung, ML-
dc.contributor.authorKo, JMY-
dc.contributor.authorDai, W-
dc.contributor.authorWong, EHW-
dc.contributor.authorKwong, D-
dc.contributor.authorNg, WT-
dc.contributor.authorLee, A-
dc.contributor.authorNgan, RK-
dc.contributor.authorYau, CC-
dc.contributor.authorTung, S-
dc.date.accessioned2014-07-07T08:26:49Z-
dc.date.available2014-07-07T08:26:49Z-
dc.date.issued2014-
dc.identifier.citationThe 105th Annual Meeting of the American Association for Cancer Research (AACR 2014), San Diego, CA., 5-9 April 2014. In Cancer Research, 2014, v. 74 n. 19 suppl., abstract no. 4148-
dc.identifier.issn0008-5472-
dc.identifier.urihttp://hdl.handle.net/10722/198639-
dc.descriptionEpidemiology - Poster Presentations - Proffered Abstracts - Poster Presentations - Molecular and Genetic Epidemiology of Lung, Head and Neck, and Gastrointestinal Cancers: abstract no. 4148-
dc.descriptionThis journal suppl. entitled: Proceedings: AACR Annual Meeting 2014; April 5-9, 2014; San Diego, CA-
dc.description.abstractBACKGROUND AND AIMS: The genetic etiology of NPC and mechanisms for inherited susceptibility remain unclear. Only modest low-penetrance effects of cancer-predisposing common variant SNPs were previously identified in the few large-scale NPC association studies reported. Most NPC association studies focused on single or limited candidate genes with modest sample sizes. Systematic and comprehensive study designs for evaluation of higher order gene-gene interactions are scanty. A large-scale NPC case-control SNP association study was performed to examine the genetic risk factors for NPC development. In order to elucidate the ...-
dc.languageeng-
dc.publisherAmerican Association for Cancer Research. The Journal's web site is located at http://cancerres.aacrjournals.org/-
dc.relation.ispartofCancer Research-
dc.rights©2014 American Association for Cancer Research.-
dc.subjectMedical sciences-
dc.subjectOncology-
dc.titleAdverse effects of TERT-CLPTM1L and double-strand breaks repair contribute to risk for nasopharyngeal carcinoma-
dc.title.alternativeAdverse effects if TERT-CLPTM1L and double-strand breaks repair contribute to risk for NPC-
dc.typeConference_Paper-
dc.identifier.emailLung, ML: mlilung@hku.hk-
dc.identifier.emailKo, JMY: joko@hku.hk-
dc.identifier.emailDai, W: weidai2@hku.hk-
dc.identifier.emailWong, EHW: elibe@hku.hk-
dc.identifier.emailKwong, D: dlwkwong@hku.hk-
dc.identifier.authorityLung, ML=rp00300-
dc.identifier.authorityKo, JMY=rp02011-
dc.identifier.authorityKwong, D=rp00414-
dc.description.naturepostprint-
dc.identifier.doi10.1158/1538-7445.AM2014-4148-
dc.identifier.hkuros229927-
dc.identifier.volume74-
dc.identifier.issue19 suppl.-
dc.identifier.spageabstract no. 4148-
dc.identifier.epageabstract no. 4148-
dc.identifier.isiWOS:000349910202129-
dc.publisher.placeUnited States-
dc.identifier.issnl0008-5472-

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